[The analgesia effect and plasma corticosterone reaction during stress in adult rats treated neonatally with monosodium glutamate].

Chen Ql、Jiehui Li、Liu Hc、GU Fu-kang、Yin Qz

1994PubMedNeuroscience被引 1

摘要

In the present investigation, it was found that the number of beta-endorphin (beta-END)-immunoreactive neurons of hypothalamic arcuate nucleus (ARC) in MSG-treated rats decreased by 60.7%. No significant differences in the baseline pain threshold and the baseline corticosterone (CS) level were observed between the MSG-treated group and the control group. After electrical foot shock for 30 min, the analgesia effect was decreased significantly (P < 0.001) in MSG-treated rats, while the CS levels of both MSG-treated rats and control rats were increased remarked (P < 0.01). The results suggest that the beta-END neurons of ARC were involved in analgesia, but not in CS reaction during stress.

引用本文（GB/T 7714）

Chen Ql, Jiehui Li, Liu Hc, 等. [The analgesia effect and plasma corticosterone reaction during stress in adult rats treated neonatally with monosodium glutamate].[J]. PubMed, 1994.

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